The Journal of Bone and Joint Surgery (American). 2005;87:639-650.
doi:10.2106/JBJS.D.02371
© 2005 The Journal of Bone and Joint Surgery, Inc.
Posterior Shoulder Dislocations and Fracture-Dislocations
C. Michael Robinson, BMedSci, FRCSEd(Orth)1 and
Joseph Aderinto, FRCSEd1
1 The Royal Infirmary of Edinburgh, Old Dalkeith Road, Edinburgh EH16 4SU,
United Kingdom. E-mail address for C.M. Robinson:
c.mike.robinson{at}ed.ac.uk
Investigation performed at The Shoulder Injury Clinic, The Royal
Infirmary of Edinburgh, Edinburgh, United Kingdom
The authors did not receive grants or outside funding in support of their
research or preparation of this manuscript. They did not receive payments or
other benefits or a commitment or agreement to provide such benefits from a
commercial entity. No commercial entity paid or directed, or agreed to pay or
direct, any benefits to any research fund, foundation, educational
institution, or other charitable or nonprofit organization with which the
authors are affiliated or associated.
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Abstract
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Posterior shoulder dislocations and fracture-dislocations are uncommon
injuries that most often occur during seizures or as a result of high-energy
trauma. Despite advances in imaging, they are frequently diagnosed late.
Detection is facilitated by heightened clinical suspicion of the injury in
high-risk individuals together with appropriate radiographic
investigation.
A wide variety of operative techniques, ranging from simple closed
reduction to soft-tissue and bone stabilization procedures to prosthetic
arthroplasty, are available to treat these injuries. Selection of the most
appropriate treatment option is complex and multifactorial. Because of the
rarity of these injuries, evidence-based treatment protocols are difficult to
devise.
Good functional outcomes are associated with early detection and treatment
of isolated posterior dislocations that are associated with a small osseous
defect and are stable following closed reduction. Poor prognostic factors
include late diagnosis, a large anterior defect in the humeral head, deformity
or arthrosis of the humeral head, an associated fracture of the proximal part
of the humerus, and the need for an arthroplasty.
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Introduction
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Posterior shoulder instability comprises a spectrum of disorders, which
include first-time traumatic dislocation (complete dissociation of the humeral
head from the glenoid) and recurrent dislocation or subluxation (symptomatic,
excessive translation of the humeral head on the glenoid). These conditions
are uncommon but are more challenging to treat than are their anterior
counterparts, and few surgeons encounter them with sufficient frequency in
their clinical practice to be confident about their assessment and treatment.
As a consequence, delays in diagnosis, errors in treatment, and protracted
morbidity frequently occur. Their rarity makes it difficult to devise
evidence-based management strategies or to obtain substantive proof of the
benefits of newer treatment methods. This review provides an overview of the
current knowledge regarding the diagnosis, classification, assessment, and
treatment of posterior shoulder dislocations and fracture-dislocations.
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Spectrum of Posterior Shoulder Instability
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First-time traumatic posterior dislocations include so-called simple
dislocations, which are usually accompanied by an anterior osteochondral
impression fracture of the humeral head, and true fracture-dislocations, which
are accompanied by more complex fractures of the tuberosities and/or proximal
part of the humerus. Recurrent episodes of posterior shoulder subluxation or
dislocation may occur following reduction of a first-time traumatic posterior
dislocation. However, symptomatic recurrent posterior instability most
commonly presents with repeated episodes of subluxation, with or without a
history of shoulder injury. The etiology of recurrent posterior instability is
complex and multifactorial and currently defies rigid classification. This
condition is the subject of a review that is to be published in the next issue
of The Journal.
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Epidemiology
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Although traumatic posterior dislocations account for <3% of all
shoulder dislocations (forty-one of 1491 dislocations in four
series1-4),
it is difficult to assess their prevalence accurately because many cases
remain clinically undetected. Many of these injuries are
fracture-dislocations1,5,6,
but only a small minority of fractures of the proximal part of the humerus
have a concomitant posterior
dislocation7,8.
Traditionally, most posterior dislocations have been associated with
epileptic seizures, high-energy trauma, electrocution, or electroconvulsive
therapy6,9,10
(although the latter two causes are now extremely
rare11). In the
absence of trauma, posterior fracture-dislocation (unilateral or bilateral) is
virtually pathognomonic of a
seizure12,13.
The rising prevalence of diabetes mellitus and of alcohol and drug dependency
has led to a greater proportion of dislocations occurring during seizures as a
result of
hypoglycemia14 or
drug
withdrawal15.
The majority of posterior dislocations occur in men between thirty-five and
fifty-five years of age, and 15% are bilateral (twenty-four of 164 patients
with posterior dislocation in five series had the condition
bilaterally1,6,16-18).
The reason for the higher prevalence in men is unclear, although they are more
commonly injured in motor-vehicle accidents and when playing sports.
Furthermore, their more muscular habitus may increase their risk of shoulder
injury during a seizure.
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Pathogenesis
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Although the humeral head and, to a lesser extent, the glenoid are both
normally retroverted with respect to their long axes, the scapula is
protracted on the chest
wall19.
Consequently, in its normal position of function, the shoulder is protected
from posterior dislocation by the strong buttressing action of the posterior
aspect of the
glenoid20,21.
Additional static stabilization is provided by the posterior capsulolabral
complex and the posterior band of the inferior glenohumeral
ligament22, while
dynamic stability is provided by the rotator cuff and the shoulder girdle
muscles.
Excessive posterior translation is also prevented by anterior constraints
that contribute to capsuloligamentous
stability23-25.
These include the rotator interval
capsule26, the
superior and middle glenohumeral and coracohumeral ligaments, and the
subscapularis
tendon23,24,27,28.
The relative contribution of these structures to stability varies with the
position of the
shoulder25,27.
Stability also depends on coordinated glenohumeral and scapulothoracic
movements29.
Traumatic posterior dislocation is possible during a high-energy injury if
an axial force is applied with the shoulder in the unstable position of
internal rotation, forward elevation, and adduction
(Fig.
1)21.
Traumatic dislocation may also be sustained as a result of sustained
contraction of the internal rotators during a seizure that occurs with the arm
at the
side30,31.
During posterior dislocation, an osteochondral impression fracture (also
termed an encoche fracture or a reverse Hill-Sachs
lesion)2 is produced
as the anterior aspect of the humeral head impacts on the posterior aspect of
the glenoid (Fig.
2-A)32.
If the shoulder is not relocated at an early stage, the head defect enlarges
and becomes corticated as a result of the grinding effect of rotational
movements. With prolonged dislocation, secondary deformity of the articular
cartilage of the humeral head may develop. This "ping-pong-ball"
effect33,34
is due to resorption of the subchondral bone of the humeral head when it is
not subjected to physiologic loading, which eventually progresses to secondary
osteoarthrosis6,18,33,35.

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Fig. 1 The shoulder is most at risk for posterior dislocation when it is axially
loaded with the arm in forward elevation, adduction, and internal rotation
during a high-energy injury. That position is also the one in which patients
with recurrent subluxation of the shoulder characteristically experience
instability symptoms. (Printed with permission of Jennifer Fairman, Fairman
Studios, Waltham, Massachusetts.)
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Fig. 2A and Fig. 2B Three-dimensional computerized tomography reconstructions of a simple
posterior dislocation (Fig. 2-A) and a posterior fracture-dislocation (Fig.
2-B) with an anatomic neck (HH) fracture and combined greater (GT) and lesser
(LT) tuberosity "shield" fragments. G = glenoid.
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Acute dislocation may produce an injury to the posterior stabilizers, which
can present as capsulolabral tears or
avulsions22,27,36-38,
glenoid rim
fractures39,40,
or rotator cuff
tears22,27,41.
There is some evidence to suggest that posterior capsular tears will heal
spontaneously following relocation of the
shoulder40,42.
If the force producing a posterior dislocation is severe or if the proximal
part of the humerus is osteoporotic, the impression fracture may propagate to
produce fractures of the tuberosities and/or proximal part of the humerus,
most commonly through the anatomic neck
(Fig. 2-B). The humeral head
has a segmental blood supply, mainly derived from the ascending branch of the
anterior circumflex humeral
artery43,44.
There is consequently a risk of osteonecrosis after fractures of the anatomic
neck, although if the fracture extends below the articular surface medially,
the head may be perfused by intact posteromedial
vessels45.
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Classification
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Because of the rarity of posterior dislocation and the complexity and
unpredictable outcome of its treatment, a satisfactory classification of this
injury has not been devised. Dislocations with a concomitant fracture of the
anatomic neck or tuberosities (fracture-dislocations) should be considered
separately from simple dislocations, which should include those associated
with an impression fracture of the humeral head
(Figs. 2-A and 2-B).
Simple Dislocations (without Associated Fracture of the Tuberosities or the Proximal Part of the Humerus)
These injuries have usually been classified according to the timing of the
diagnosis, with those diagnosed immediately or within the first six weeks
after the injury traditionally classified separately from those that are
detected
later2,16,33,46.
Dislocations that present late have previously been termed
"chronic"33,47,
"neglected"48,
or
"locked"6,
although these terms are misleading and probably best avoided.
In reality, the temporal delay in diagnosis is a continuum, during which
the humeral head defect becomes progressively larger and the shoulder becomes
more difficult to reduce, until it eventually becomes deformed and arthritic.
As these factors most commonly influence treatment, it is preferable to use
them for subclassification rather than basing the classification solely on the
timing of the diagnosis. The treatment and prognosis of dislocations with a
head defect measuring <40% to 50% of the articular surface is usually
considered to be different from those with a larger defect and those with
deformity or arthritic change in the remainder of the humeral
head6,17,18,49.
The subclassification used in this review is based on this distinction.
Fracture-Dislocations (Dislocations with Fractures of the Tuberosities and/or Proximal Part of the Humerus)
Posterior fracture-dislocations are included in most shoulder fracture
classifications7,32,50.
The three most commonly encountered fracture patterns are two-part fractures
of the lesser tuberosity (Group V in the Neer system and not classified in the
AO
system)48,51-53,
two-part fractures of the anatomic neck (Group VI in the Neer system and Type
C3.1 in the AO
system)53,54,
and complex three-part and four-part fractures (Group VI in the Neer system
and Types B3.3, C3.2, and C3.3 in the AO
system)7,49,54-58
(Fig. 2-B).
The important distinction between fracture-dislocations, in which the
humeral head is locked on the posterior aspect of the glenoid, and
fracture-subluxations is unclear in most classification
systems59. The
latter are typically osteoporotic, multipart fractures in the elderly, with
the humeral head subluxated posteriorly but not locked on the glenoid rim or
extruded60,61.
Fracture-subluxations should be considered with other Neer Group-III and IV,
three and four-part fractures. These injuries are not discussed in detail in
this review.
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Assessment and Diagnosis
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Delays in the diagnosis of posterior dislocations are common: it is
estimated that up to 50% are missed when the patient first seeks medical
consultation40,46,62.
Common causes for delay include late presentation by the patient, the
physician's failure to suspect the diagnosis on examination, and inadequate
radiographic
investigation63.
Dislocations are more commonly missed in the
elderly5, in
patients with concomitant
fractures64,65,
and in those with multiple injuries or other injuries in the same
limb1. The key to
diagnosis lies in maintaining a high index of clinical suspicion and
performing appropriate radiographic investigations.
Clinical Assessment
An acute posterior dislocation is characteristically painful, but affected
patients may have reduced nociception following a seizure, especially in
instances of drug or alcohol intoxication. Although the absolute risk of
dislocation during a seizure is
small13,31,
the diagnosis must be excluded if shoulder pain is present following such an
episode. In the absence of a history of epilepsy, investigation is required to
exclude underlying causes, such as an intracranial mass lesion or metabolic
abnormality66,67.
The dislocated shoulder is typically held in internal rotation, and the
most consistent finding is a mechanical block to external rotation, caused by
engagement of the anterior humeral head defect on the posterior aspect of the
glenoid2. The
absence of pain on rotational shoulder movements is suggestive of a chronic
dislocation, but the exact age of a dislocation may be difficult to define,
particularly if a patient with unstable epilepsy has had shoulder pain for
some time. Concomitant tears of the rotator cuff and neurovascular injury are
uncommon, but those diagnoses should be
excluded39-41.
Patients who sustain a posterior dislocation during a seizure often have
medical and psychosocial problems, which may be related to the conditions that
caused the seizure, such as alcoholism or drug dependency, or to noncompliance
with their treatment with anticonvulsant
medication68,69.
Treatment of unstable medical problems may be appropriate prior to operative
intervention. A psychological assessment may also be useful in determining the
patient's likely receptivity and compliance with treatment.
Imaging Studies
Many radiographic signs of posterior dislocation on standard
anteroposterior radiographs have been
described70,71.
However, these signs are unreliable, and an axillary radiograph is essential
to confirm the diagnosis. Standard axial radiographs are difficult to make
because the patient usually cannot abduct the arm. Apical
oblique72,
Velpeau73, or
modified axial
radiographs74 are
therefore preferable, as they can be made with the arm in a sling (Figs.
3-A,
3-B, and 3-C). Ultrasound has
been used to detect posterior dislocation, but it requires specialist training
and cannot accurately demonstrate the extent of osseous
pathology75.

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Fig. 3-A The configuration of the x-ray gantry and cassette used to make a modified
axial radiograph of the shoulder.
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Fig. 3-B Conventional anteroposterior radiograph of a shoulder with a posterior
dislocation, which is not evident on this view. Fig. 3-C The posterior
dislocation is clearly evident on the modified axial radiograph of the same
shoulder.
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Additional imaging is essential in the evaluation of posterior dislocations
prior to treatment. Computerized tomography provides good definition of the
osseous lesions that determine
treatment48,68.
It can detect radiographically occult anatomical neck
fractures76, and it
enables an accurate assessment of the size of the humeral head defect and the
shape and degree of degenerative change in the remainder of the humeral
head48,68,75.
Occasionally, assessment of the size and degree of cortication of the humeral
head defect can help to determine whether a dislocation occurred recently or
has been present for a longer period of time. Three-dimensional
reconstructions are available with modern spiral scanners, which can be useful
in planning operative reconstruction (Figs.
2-A and 2-B). Magnetic resonance imaging may also provide useful
information regarding the condition of the rotator cuff and the ligamentous
soft-tissue restraints of the shoulder.
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Treatment
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If the interval between the dislocation and the diagnosis has been
prolonged, it is sometimes preferable not to treat the dislocation. This is
particularly true if the patient is frail, elderly, or demented; has unstable
epilepsy; or has only minor functional
disability2,6,75.
After physiotherapy, the final degree of functional incapacity is often
minimal, given such patients' limited functional
expectations77.
Active treatment is indicated for all remaining patients, with the aim of
achieving a congruously relocated, stable, functional, and pain-free shoulder.
A wide variety of reconstructive techniques, ranging from simple closed
reduction to the more complex humeral head salvage and head-replacement
procedures, may be required to treat these complex injuries.
Dislocations with Involvement of Less Than Forty to Fifty Percent of the Humeral Head Articular Surface and No Deformity or Degenerative Changes
This pattern of injury is usually recognized in dislocations diagnosed
immediately or within the first six weeks postinjury. A posterior dislocation
may occasionally reduce spontaneously or, rarely, the patient may have
relocated the shoulder prior to seeking medical advice. However, this scenario
is much less common than it is following anterior dislocation, and most
first-time posterior dislocations require reduction under medical
supervision.
Treatment consists of closed relocation of the shoulder followed by
assessment of its stability. This is difficult in a conscious, sedated
patient, so posterior dislocations should be reduced with the patient under
general anesthesia. Ancillary stabilization techniques may be required if the
reduction is unstable.
Attempted closed reduction is indicated for dislocations with a small
humeral head defect that are diagnosed immediately or within the first six
weeks after the
injury15,46.
The impression fracture is disimpacted and is cleared from the posterior
glenoid lip with use of gentle manipulation while the arm is flexed 90°
and adducted1.
External rotation at this stage will relocate the shoulder, but it should not
be attempted before the defect has been fully disengaged, as there is a risk
of fracturing the humerus.
Open reduction is indicated if attempted closed relocation is unsuccessful,
which is typical if the diagnosis has been delayed and/or there is a large
humeral head defect. The deltopectoral approach is most commonly
used2,6,
but it provides limited access to the posterior aspect of the glenoid for
relocation of the humeral
head78. A separate
posterior approach is sometimes required if the head is
irreducible79.
The deltoid-splitting, superior subacromial approach is an alternative
favored by some
authors78,80
as it provides more direct access to the dislocated humeral head. Most
reconstructive procedures can be performed through this
approach61,
although there is a risk of postoperative detachment of the deltoid from the
acromion and injury to the axillary nerve if the split is continued too
distally. Irrespective of the approach that is used, following arthrotomy at
the rotator interval and prior to relocation, the impression fracture should
be disengaged from the glenoid rim under direct vision.
Following reduction, stability is assessed by internal rotation of the
shoulder to establish the point at which redislocation occurs. The degree of
instability is usually determined by the size of the anterior humeral head
defect, as it reengages with the posterior aspect of the glenoid rim, although
instability can also be produced by posterior capsular injury. If the
reduction is stable throughout a functional range of rotation (usually in a
patient with a dislocation accompanied by a small humeral head defect
occupying <25% of the articular
surface18,40),
immobilization of the shoulder in neutral or external rotation for four weeks
is recommended (Fig.
4)40.

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Fig. 4 Following reduction of a posterior dislocation, the shoulder is in its most
stable position when it is in neutral forward elevation and external rotation
because the anterior humeral head defect is prevented from reengaging with the
posterior aspect of the glenoid. This is the generally preferred position of
immobilization following relocation of the shoulder to prevent acute
redislocation. (Printed with permission of Jennifer Fairman, Fairman Studios,
Waltham, Massachusetts.)
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If the shoulder redislocates as it is internally rotated, there are two
choices for treatment: an adjunctive stabilization procedure or immobilization
in a more stable position of external rotation
(Fig. 4), which moves the area
of the anterior humeral head defect away from the posterior aspect of the
glenoid
rim75,81.
Although these two options have not been directly compared, operative
stabilization is being increasingly recommended because of the shortcomings of
immobilization, which include shoulder stiffness, acute redislocation, and
recurrent
instability2,81,82.
Furthermore, splinting of the arm in external rotation is difficult to
maintain, unless a permanent shoulder spica is
applied20,81.
The aim of adjunctive stabilization procedures is to restore stability
throughout a functional range of movement, thereby preventing an acute
redislocation. Transposition of the subscapularis tendon into the humeral head
defect, either alone (the McLaughlin procedure,
Fig.
5)2 or
in continuity with the osteotomized lesser tuberosity when there is a larger
defect (the Neer modification of the McLaughlin
technique)6,83,
has been widely
used48,75,84.
Of the two variations, the latter is preferable when there is an associated
fracture of the lesser
tuberosity52.
Rotational osteotomy of the proximal part of the humerus has also been
described85, but it
is not widely used because of its technical difficulty and the risk of
devascularizing the humeral
head17.

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Fig. 5 Schematic drawing of the McLaughlin procedure. (Printed with permission of
Jennifer Fairman, Fairman Studios, Waltham, Massachusetts.)
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These nonanatomic techniques restore stability when they are used to treat
smaller impression fractures, but they are less successful when the head
defect occupies 33% to 50% of the articular
surface6,18,40,52,68,86.
Additionally, there is a risk of subscapularis dysfunction, leading to
weakness of internal rotation or contracture, which may complicate later joint
replacement40.
Disimpaction, elevation, and autogenous bone-grafting of the depressed
osteochondral humeral head fracture has more recently been used to treat
residual instability in patients in whom the dislocation had been diagnosed
within the first two weeks after the injury
(Fig.
6)40,68,87,88.
When the remaining articular surface is not osteoporotic, deformed, or
arthritic, a sculpted osteochondral humeral or femoral head allograft may be
preferable for larger defects, occupying up to half of the articular surface
(Fig.
7)40,47,68.
Only brief reports of these newer anatomic techniques are
available40,68,
although the functional results of allografting are considered to be similar
to those of subscapularis
transfer40.

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Fig. 6 Schematic representation of acute elevation, bone-grafting, and internal
fixation of a small anterior osteochondral fracture of the humeral head. A
channel is created through the humeral head to gain access to the impaction
fracture cavity. The cavity is then filled with bone graft, which is impacted
to elevate the depressed segment of articular cartilage. (Printed with
permission of Jennifer Fairman, Fairman Studios, Waltham, Massachusetts.)
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Fig. 7 Schematic diagram of the use of a solid osteochondral allograft to fill a
larger anterior humeral head defect. (Printed with permission of Jennifer
Fairman, Fairman Studios, Waltham, Massachusetts.)
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Although there are no evidence-based guidelines for treatment, we currently
recommend disimpaction and bonegrafting for small defects (<25% of the
humeral head surface) that are recognized within the first two weeks after
injury. Subscapularis transfer is used for smaller defects diagnosed more than
two weeks after injury. Larger lesions, occupying up to 40% to 50% of the
articular surface, are filled with a structural allograft, irrespective of the
duration of the dislocation. Stability should be reassessed after the
adjunctive procedure. Inadequate restoration of stability may be due to a
posterior capsulolabral disruption or a displaced posterior glenoid
fracture69, which
may require
stabilization68.
Dislocations with Involvement of More Than Forty to Fifty Percent of the Humeral Head Surface or with Deformity or Secondary Osteoarthrosis of the Humeral Head
This pattern of injury is most commonly present in patients for whom the
diagnosis had been delayed or in older patients who have substantial softening
of the humeral head due to osteoporosis. Open reduction and allograft
stabilization of the humeral head has been used to treat carefully selected,
younger patients with a defect of >50% of the head surface and preserved
sphericity of the humeral
head40. However,
arthroplasty is more commonly used to treat dislocations associated with a
large humeral head
defect6,18
or those associated with deformity (a "ping-pong-ball" head) or
secondary osteoarthrosis of the articular
surface18,33.
Both hemiarthroplasty and total shoulder
replacement18,40,46,79,89
have been used, although they have never been directly compared, to our
knowledge. Some authors have recommended use of total shoulder replacement
only when there is reciprocal arthritic change in the
glenoid6,18,46,
whereas others use it routinely, claiming that it provides better pain relief
and restoration of the
anatomy40,79.
A recognized technical problem of arthroplasty is the predisposition to
posterior subluxation or dislocation of the prosthesis due to
anterior-inferior capsular and subscapularis contractures. The prosthesis has
been inserted in a neutral or relatively anteverted orientation to reduce this
risk6,46,89,
but capsular release together with normal prosthetic retroversion is now
favored by many
authors18,40,75.
Rarely, when there is severe humeral head deformity, an arthroplasty,
arthrodesis, or excision of the humeral head (Jones procedure) may be
considered16,17,33.
There is no evidence with which to delineate the relative indications for
these procedures, but an arthroplasty is usually preferable if it is
technically feasible, as the functional outcome is likely to be better.
Fracture-Dislocations
Dislocations with an undisplaced lesser tuberosity fracture can be treated
in the same manner as simple
dislocations57, but
open reduction and internal fixation is recommended if there is displacement.
The tuberosity may be fixed either anatomically or into the base of the
humeral head defect if the shoulder is unstable. Two, three, and four-part
fractures involving the anatomic neck are treated either with reduction and
internal fixation or with arthroplasty. The treatment is determined by the age
and medical status of the patient and the degree of devascularization and
fragmentation of the humeral head and
tuberosities57.
Arthroplasty is generally favored for multipart fractures in the
elderly18,57,79.
In addition to the technical problems encountered when an arthroplasty is used
to treat a simple dislocation, it may be difficult to judge the appropriate
prosthetic soft-tissue tensioning and to obtain stable fixation of the
fractured tuberosities (Fig.
8-A)57,90,91.


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Fig. 8-A Severely comminuted multipart posterior fracture-dislocations in the
elderly are usually treated with hemiarthroplasty. In this patient, the
prosthesis was well centered on the glenoid (with the center of the humeral
head prosthesis displaced only 3 mm from the center of the glenoid) and the
tuberosities healed without migration, leading to a satisfactory functional
outcome. Fig. 8-B In younger individuals with a posterior
fracture-dislocation, head salvage, typically with open reduction and plate
fixation, is attempted whenever possible.
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Head-salvaging reconstruction is usually attempted in younger adults with
good bone stock. While closed reduction techniques have been
described1,54,92,
open reduction and internal fixation is usually preferred because it reduces
the risk of iatrogenic fracture
displacement93.
Temporary transfixation wires may prevent displacement of undisplaced neck
fractures during open
relocation93.
The type of internal fixation used is determined by the complexity of the
fracture. Two-part fractures may be stabilized by simple interfragmentary
screws34, whereas
multipart fractures require more complex reconstruction with use of buttress
plates, tension bands, or intramedullary fixation
(Fig.
8-B)8,94-96.
Stability should be assessed following internal fixation, as adjunctive
procedures may be required if there is residual instability.
Neglected fracture-dislocations may be difficult to reduce operatively, and
malunion of the proximal part of the humerus makes reconstructive procedures
technically demanding. If an arthroplasty is attempted, it is best to avoid
osteotomy and realignment of the tuberosities, as this can be associated with
nonunion, secondary displacement, and a poor functional
outcome40,75,97.
In many cases, a joint excision or fusion may be the only technically feasible
option18,33.
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Outcome and Complications of Posterior Dislocation
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As a result of the rarity of posterior dislocations, most of the published
literature consists of Level-IV (case-series) and Level-V (expert opinion)
studies, often compiled retrospectively over many years. Comparative studies
of different treatment methods, which would help to resolve areas of
controversy, are lacking. Furthermore, because a wide variety of subjective
and objective methods has been used to assess functional outcome, it is
impossible to compare different studies. The most consistently recorded
outcome measure is the occurrence of complications. Although it is generally
accepted that the risk of complications is high, their exact prevalence is
often difficult to quantify because of variable recording and differences in
both case-mix and treatment philosophies in published studies.
In general, a less favorable functional outcome is anticipated and the risk
of all complications is higher when the dislocation is diagnosed
late1,2,5,18,86,
when the humeral head impression defect is
large6, when
secondary humeral head deformity and osteoarthrosis are
present2,6,
or when there is a concomitant proximal humeral
fracture17,18.
Outcomes have also been reported to be less satisfactory when closed reduction
alone is not
possible5,6,18
or when an arthroplasty is required to treat the
dislocation18,33.
The following complications have been specifically associated with posterior
dislocation.
Acute Redislocation
This complication may occur following closed relocation or be due to
failure of an adjunctive surgical stabilization procedure. Reported
redislocation rates following nonoperative treatment have varied widely.
Detenbeck51 treated
ten shoulders and reported no redislocations, whereas Wilson and
McKeever1,
Dimon82, and
Roberts and
Wickstrom39
reported redislocations in four of eleven shoulders, one of three shoulders,
and nine of twenty-four shoulders, respectively. Redislocations following
nonoperative treatment have often been recognized late and have been treated
nonoperatively, with open reduction and adjunctive surgical stabilization, or
with an arthroplasty.
Redislocation following adjunctive stabilization with the McLaughlin
procedure was reported in five
patients6. All of
those patients were referred from outside the authors' institution, and it
appeared that the procedure had been used to treat dislocations associated
with larger humeral head defects. The patients were subsequently treated
either nonoperatively or with an arthroplasty.
Recurrent episodes of posterior instability may occur following an initial
dislocation and are best considered within the spectrum of recurrent posterior
instability. These will be discussed in the subsequent review concerning
recurrent posterior shoulder instability.
Osteonecrosis
Osteonecrosis of the humeral head has been reported following simple
dislocation47, but
it is more frequently encountered following internal fixation of an anatomic
neck
fracture-dislocation17,93.
The risk of osteonecrosis increases with the degree of fracture displacement
and the extent of involvement of the
tuberosities57.
However, following anatomic reconstruction, the head may continue to be
perfused by intact posteromedial
vessels45 or,
alternatively, revascularization by creeping substitution may
occur98,99.
Osteonecrosis may be associated with satisfactory function if an anatomic
reconstruction was previously
achieved95,100.
Symptomatic patients are usually treated with an
arthroplasty57.
Posttraumatic Osteoarthrosis
Posttraumatic degeneration of the glenohumeral joint is relatively uncommon
after posterior dislocation, but when it occurs the severity of the arthrosis
is usually worse than that following anterior
dislocation35. If
symptoms are severe enough to warrant treatment, a shoulder arthroplasty is
usually performed.
Joint Stiffness and Functional Incapacity
Persistent shoulder stiffness and functional incapacity after a simple
dislocation are associated with a delay in the diagnosis; deformity,
osteoarthrosis, or osteonecrosis of the humeral head; a tear of the rotator
cuff41; a
complication of an ancillary stabilization
procedure6; or
treatment with an arthroplasty, excision, or
arthrodesis6,18.
The treatment of stiffness in these patients should be directed toward the
underlying cause. However, the cause of stiffness is often obscure and is
assumed to be due to periarticular fibrosis and capsular contracture. This may
respond to manipulation and an intensive physical therapy
regimen57, but it
tends to be refractory to treatment in most patients.
The functional results of reduction and internal fixation of a
fracture-dislocation are satisfactory in patients in whom complications do not
develop18,95.
However, joint stiffness may occur if a nonunion or malunion of the humeral
head
develops57,98,100.
Closed manipulation is not advised for the treatment of persistent stiffness,
as there is an unacceptable risk of
refracture57;
treatment should be directed toward correcting the underlying cause.
Stiffness and a poor functional outcome are also common when an
arthroplasty is used to treat a
fracture-dislocation79,90,91,101,102.
The outcome is often comparable with that of an arthrodesis, with the patient
having pain-free scapulothoracic movement
only91. An
arthroplasty is more likely to result in poor function in the elderly and in
patients with malunion or nonunion of the
tuberosities90,
glenoid
arthrosis6,18,
or subluxation of the
prosthesis18,91.
The joint stiffness is often refractory to treatment, and there have been no
reports of the use of revision arthroplasty to treat this complication, to our
knowledge.
 |
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