The Journal of Bone and Joint Surgery (American). 2009;91:1758-1776.
doi:10.2106/JBJS.H.01348
© 2009 The Journal of Bone and Joint Surgery, Inc.
Blount Disease
Sanjeev Sabharwal, MD1
1 Department of Orthopedics, University of Medicine and Dentistry of New Jersey, New Jersey Medical School, Doctor's Office Center, 90 Bergen Street, Suite 7300, Newark, NJ 07103. E-mail address: sabharsa{at}umdnj.edu
Disclosure: The author did not receive any outside funding or grants in support of his research for or preparation of this work. Neither he nor a member of his immediate family received payments or other benefits or a commitment or agreement to provide such benefits from a commercial entity. A commercial entity (Smith and Nephew) paid or directed in any one year, or agreed to pay or direct, benefits in excess of $10,000 to a research fund, foundation, division, center, clinical practice, or other charitable or nonprofit organization with which the author, or a member of his immediate family, is affiliated or associated.
Two clinically distinct forms of Blount disease (early-onset and late-onset), based on whether the lower-limb deformity develops before or after the age of four years, have been described.
Although the etiology of Blount disease may be multifactorial, the strong association with childhood obesity suggests a mechanical basis.
A comprehensive analysis of multiplanar deformities in the lower extremity reveals tibial varus, procurvatum, and internal torsion along with limb shortening. Additionally, distal femoral varus is commonly noted in the late-onset form.
When a patient has early-onset disease, a realignment tibial osteotomy before the age of four years decreases the risk of recurrent deformity.
Gradual correction with distraction osteogenesis is an effective means of achieving an accurate multiplanar correction, especially in patients with late-onset disease.

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